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Renal response to metabolic acidosis: role of mRNA stabilization
Faculty
Medicine
Year:
1900
Type of Publication:
ZU Hosted
Pages:
Authors:
hend Mohamed Mohamed Ibraheim
Staff Zu Site
Abstract In Staff Site
Journal:
لايوجد
Volume:
Keywords :
Renal response , metabolic acidosis: role , mRNA
Abstract:
Not Available
Author Related Publications
hend Mohamed Mohamed Ibraheim, "Role of deadenylation and AUF1 binding in the pH-responsive stabilization of glutaminase mRNA. Am J Physiol Renal Physiol.: (porterJM, Ibrahim H, Taylor L, Curthoys NP(2006", لايوجد, 1900
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hend Mohamed Mohamed Ibraheim, "Complexity and species variation of the kidney-type glutaminase gene", لايوجد, 1900
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hend Mohamed Mohamed Ibraheim, "Complexity and species variation of the kidney-type glutaminase gene", لايوجد, 1900
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hend Mohamed Mohamed Ibraheim, "RNA recognition by 3?-to-5? exonucleases: The substrate perspective", لايوجد, 1900
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hend Mohamed Mohamed Ibraheim, "Role of deadenylation and AUF1 binding in the pH-responsive stabilization of glutaminase mRNA", لايوجد, 1900
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Department Related Publications
Nourhanne Abdullah Saeed, "MicroRNA-199b Expression Level and Coliform Count in Irritable Bowel Syndrome", IUBMB Life, 2016
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Amal Mohamed Hussin Haassan, "Endothelial Nitric Oxide Synthase (enos) -786T>C and +894G>T Gene Polymorphisms and Their Association With Coronary Artery Disease Risk Factors in Type 2 Diabetic Egyptian Patients", ZAGAZIG UNIVERSITY MEDICAL JOURNAL, 2012
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Rehab Ahmed Karam Abdelfattah, "Diagnostic and prognostic significance of serum miRNA‐146‐a expression in Egyptian children with sepsis in pediatric intensive care unit", willey, 2019
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Samia Hussein Aly Mohammed, "The Effect of Intermittent Fasting and Vitamin Supplement on Cognitive Functions in Type 2 Diabetes Mellitus", Springer Nature, 2025
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Doaa Mahmoud AbdulHameed Saleh, "CXCR 4, PAX5, and OCT2 Immunohistochemical Expressions in Diffuse Large B-Cell Lymphoma and Their Relationship with XIST/miR-34/PAX5 Axis", Mary Ann Liebert, Inc., with operational and platform hosting provided in partnership with SAGE Publications, 2025
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